The importance of antibiotics in reducing diseases to which the body is exposed has been tested sensitivity to antibiotics and all types of bacteria isolated from various infections in the body, according to the method (Kerby Bauer, 1966), the researcher said. There is a gradual change in the types of bacteria pathogenic in their sensitivity to antibiotics, so we can not expect a consistent pattern of the sensitivity of these bacteria to antibiotics.
Staphylococcus isolates showed Staph. (34.93,41.37,41.37,58.62), respectively, while high sensitivity against Streptomycin (%) was found to be resistant to antitetracycline, 89.66)
Staph.epidermis was resistant to Neomycin (66.66%), followed by Ceftazidime, Rifampicin (50%), and was highly sensitive to Ampicillin (83.34%). Neomycin had a resistance percentage of Bacteria (% 53.84) Strepto. Pyogen, which was sensitive to Cefotaxime (92.37%), was also resistant to Ciprofloxacin (46.15%), followed by Rifampicin (30.76%). These results converge with what was reported.
Escherichia coli was resistant to Ampicillin (91.66%), followed by Ceftazidime and Tetracyclin (75.83.33) respectively, while it was sensitive to Cefotaxime (75%) while Proteus. mirabilus resistant to antimicrobial Ampicillin (83.33%) and more sensitive to anti-Neomycin, which was resistant to this antibody (25%).
While Klebsiella spp was very resistant to Cefotaxime (100%), Ampicillin (100%) and Streptomycin (5%). Corynebacterium dipthheroid (C) showed high resistance to Ampicillin, Ceftazidime, Rifampicin, Trimethprim, Streptomycin, 100) while high-sensitivity to tetracyclin, Neomycin, Ciprofloxacin, Cefotaxime and Haemophillas spp. Streptomycin, Ampicillin (80%) was highly resistant to Rifampicin Tetracyclin and Neomycin (20%).
Pseudomonas aeruginosa had high resistance to Ampicillin, Ceftazidime (71.42,78.57%) and less resistance to Cefotaxime (14.28%) than other antibiotics.
The results of the current study showed that the best antagonists in their effect on the isolates of Staph bacteria. Aureus, Staph.epidermis, was antifungal Streptomycin, while Ciprofloxacin had a 100% effect on Corynebacterium dipthheroid bacteria, while Cefotaxime was one of the best antagonists against Pseudomonas spp (85.72%).
The rest of the resistance ranged up to the rest of the antibodies in a similar manner. These ratios are close to those of local and international researchers because of their wide spectrum efficiency against isolated bacteria.
David et al. (2001) indicated increased resistance to Gram-negative bacteria, particularly intestinal family members, due to the production of plasmid-borne bactalactase enzymes. They noted that Proteus mirabilis, Escherichia coli contains a plasmid that encodes the production of large-spectrum bactalactase enzymes responsible for Resistance to cephalosporins and the production of these enzymes are important functions of the Gram-negative bacteria and resistance to this group of antibiotics.
The resistance of isolated bacteria to more than one antiviral such as Streptomycin, Ampicillin, Ceftazidime, Tetracyclin and Neomycin is due to the indiscriminate use or misuse of antibiotics due to mutations due to the repeated use of these antibiotics or the containment of bacteria on plasmid carrying genes of resistance to these antibiotics.
Staphylococcus isolates showed Staph. (34.93,41.37,41.37,58.62), respectively, while high sensitivity against Streptomycin (%) was found to be resistant to antitetracycline, 89.66)
Staph.epidermis was resistant to Neomycin (66.66%), followed by Ceftazidime, Rifampicin (50%), and was highly sensitive to Ampicillin (83.34%). Neomycin had a resistance percentage of Bacteria (% 53.84) Strepto. Pyogen, which was sensitive to Cefotaxime (92.37%), was also resistant to Ciprofloxacin (46.15%), followed by Rifampicin (30.76%). These results converge with what was reported.
Escherichia coli was resistant to Ampicillin (91.66%), followed by Ceftazidime and Tetracyclin (75.83.33) respectively, while it was sensitive to Cefotaxime (75%) while Proteus. mirabilus resistant to antimicrobial Ampicillin (83.33%) and more sensitive to anti-Neomycin, which was resistant to this antibody (25%).
While Klebsiella spp was very resistant to Cefotaxime (100%), Ampicillin (100%) and Streptomycin (5%). Corynebacterium dipthheroid (C) showed high resistance to Ampicillin, Ceftazidime, Rifampicin, Trimethprim, Streptomycin, 100) while high-sensitivity to tetracyclin, Neomycin, Ciprofloxacin, Cefotaxime and Haemophillas spp. Streptomycin, Ampicillin (80%) was highly resistant to Rifampicin Tetracyclin and Neomycin (20%).
Pseudomonas aeruginosa had high resistance to Ampicillin, Ceftazidime (71.42,78.57%) and less resistance to Cefotaxime (14.28%) than other antibiotics.
The results of the current study showed that the best antagonists in their effect on the isolates of Staph bacteria. Aureus, Staph.epidermis, was antifungal Streptomycin, while Ciprofloxacin had a 100% effect on Corynebacterium dipthheroid bacteria, while Cefotaxime was one of the best antagonists against Pseudomonas spp (85.72%).
The rest of the resistance ranged up to the rest of the antibodies in a similar manner. These ratios are close to those of local and international researchers because of their wide spectrum efficiency against isolated bacteria.
David et al. (2001) indicated increased resistance to Gram-negative bacteria, particularly intestinal family members, due to the production of plasmid-borne bactalactase enzymes. They noted that Proteus mirabilis, Escherichia coli contains a plasmid that encodes the production of large-spectrum bactalactase enzymes responsible for Resistance to cephalosporins and the production of these enzymes are important functions of the Gram-negative bacteria and resistance to this group of antibiotics.
The resistance of isolated bacteria to more than one antiviral such as Streptomycin, Ampicillin, Ceftazidime, Tetracyclin and Neomycin is due to the indiscriminate use or misuse of antibiotics due to mutations due to the repeated use of these antibiotics or the containment of bacteria on plasmid carrying genes of resistance to these antibiotics.
